Use “postprandial” in a sentence | “postprandial” example sentences
1- He complained almost ceaselessly of postprandial nausea.
2- His postprandial nap was disturbed by the arrival of the boss.
3- So a postprandial blood glucose test is important , too.
4- They occur more often in the postprandial period.
5- postprandial disruption of migrating myoelectric complex in dogs.
6- postprandial PP release was at a maximum in the untreated state.
7- After each mode of octreotide treatment, postprandial residual gall bladder volume increased.
8- Eight hours after octreotide injection, postprandial gall bladder contraction was partially restored.
9- The calcium channel blocker nicardipine decreases rectal postprandial motor activity in normal subjects.
10- The two-hour postprandial test is done exactly two hours after eating a meal.
11- Diabetics and those with postprandial blood glucose levels between 5.4-11.
12- The highest wave frequency and motility index were noted in the postprandial period.
13- This leads to lower postprandial glucose levels and less frequent episodes of hypoglycaemia.
14- During CSOI, postprandial gall bladder contraction is partially preserved during all meals.
15- Low-carbohydrate diets might seem to be a logical approach to lowering postprandial glucose.
16- Hyperglycemia induced by glucocorticosteroids is primarily an exaggeration of postprandial hyperglycemia.
17- During CSOI, postprandial CCK release was suppressed.
18- The pattern of steroid-induced hyperglycemia is an exaggeration of postprandial hyperglycemia.
19- Patients with postprandial abdominal pain may try a low-fat diet supplemented with increased protein.
20- Without treatment, postprandial gall bladder contraction was 86.2 (2.1%).
21- Therapy for corticosteroid-induced hyperglycemia should target postprandial hyperglycemia.
22- The addition of cisapride ed to a further reduction of postprandial reflux after lunch.
23- Intake of food by mouth was nearly impossible because of postprandial vomiting with pain.
24- This study indicates that octreotide injections impair postprandial gall bladder contraction for at least four hours.
25- postprandial triglyceride and retinyl ester responses to oral fat: effects of fructose.
26- In the past, postprandial hyperglycemia has been considered a risk factor associated mainly with diabetes.
27- This rapid action reduces the risk for hypoglycemic events after eating ( postprandial hypoglycemia).
28- postprandial CCK release was normal at eight hours after injection but very low during CSOI.
29- This meal was designed to include many reflux provoking foods to maximise postprandial reflux in normal subjects.
30- Dietary fiber tends to lower postprandial glucose response (Anderson and Akanji, 1991).
31- Regular or rapid-acting insulin analogues are the preferred treatment for postprandial hyperglycemia in pregnant women.
32- Also during CSOI, postprandial gall bladder contraction is partially resotred while CCK release remains low.
33- postprandial CCK release was suppressed for at least four hours after injection (Fig 3).
34- Both the quantity and the type or source of carbohydrates found in foods influence postprandial glucose levels.
35- If this type of low blood pressure occurs after eating, it is called postprandial orthostatic hypotension.
36- A nonlinear relationship was observed between coronary heart and stroke mortality with the two hour postprandial blood glucose.
37- However, the industrial nations do not seem ready to adopt a healthy habit of postprandial nap.
38- The finding that H pylori infection selectively increases G17 explains why the infection causes mainly postprandial hypergastrinaemia.
39- Patients with colorectal cancer are reported to have postprandial hypergastrinaemia which returns to normal after tumour resection.
40- Why revamp postprandial sea for a gasoline evidenced by substrates when we can fol one conveniently ourselves?
41- This combination therapy has the ability to greatly improve the regulation of prandial and postprandial blood glucose levels .
42- Two findings point to an important role of CCK in the regulation of postprandial gall bladder motility.
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